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The Internet Journal of Neurology ISSN: 1531-295X


Anterior Choroidal Artery Infarct


B. Robottom Department of Neurology, University of Maryland School of Medicine Baltimore, Maryland
J. Cabassa Department of Neurology, University of Maryland School of Medicine Baltimore, Maryland
M. Wozniak Department of Neurology, University of Maryland School of Medicine Baltimore, Maryland
SG. Reich Department of Neurology, University of Maryland School of Medicine Baltimore, Maryland

Citation:  B. Robottom, J. Cabassa, M. Wozniak, S. Reich: Anterior Choroidal Artery Infarct. The Internet Journal of Neurology. 2008 Volume 10 Number 1


Abstract


An 83 year-old cigarette smoker presented with left sided weakness. Examination revealed a left homonymous hemianopsia, left hemiparesis affecting face and arm greater than leg, and left hemihypesthesia with intact graphesthesia and stereognosis. MRI showed an area of restricted diffusion corresponding to an infarct in the anterior choroidal artery distribution (figure). Anterior choroidal artery infarcts are uncommon accounting for 2% of ischemic stroke1. They are distinguished by a unique clinical syndrome of hemiparesis, hemihypesthesia, and visual field deficits but without other cortical signs1. The most common etiologies include cardioembolism (54%), arterial embolus (17%), and small vessel disease (6%)2.

Figure: Diffusion weighted imaging (left) and corresponding apparent diffusion coefficient map (right) demonstrating acute infarction. The territory supplied by the anterior choroidal artery can include the posterior limb of the internal capsule, choroid plexus, initial segments of the optic radiations, and parts of amygdala, uncus, and globus pallidus.

References

1. Hohr JP, Timsit S. Choroidal Artery Disease. In: Barnett HJM, Mohr JP, Stein BM, Yatsu FM, editors. Stroke: Pathophysiology, Diagnosis, and Management. New York: Churchill Livingstone; 1998:503-512.
2. Leys D, Mounier-Vehier F, Lavenu I, et al. Anterior choroidal artery territory infarcts. Study of presumed mechanisms. Stroke 1994;25:837-842.

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